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| Dernière visite: il y a moins d’une minute | Nous sommes le 09 Mar 2026, 01:02 |
: Because it is "antisense," it is transcribed from the opposite strand of a well-known gene, a common feature in complex genetic regulation . Historical and Scientific Context
| Feature | TAR1a | RPM1 | RPS2 | N (Tobacco) | | :--- | :--- | :--- | :--- | :--- | | | TIR-NBS-LRR | CC-NBS-LRR | CC-NBS-LRR | TIR-NBS-LRR | | Guardee Protein | RIN4 | RIN4 | RIN4 | Helicase domain of NRIP1 | | Pathogen Effector | AvrRpt2 ( Pseudomonas ) | AvrRpm1/AvrB | AvrRpt2 | p50 helicase (TMV) | | Signaling Partners | EDS1, PAD4 | NDR1 | NDR1 | EDS1, NRG1 | | Cell Death Speed | Fast (8-12 hrs) | Very Fast (4-6 hrs) | Fast (12-16 hrs) | Slow (24-48 hrs) | : Because it is "antisense," it is transcribed
: It plays a role in how cells manage nitrogen metabolism and retrograde signaling between the mitochondria and the nucleus. Clarification: Did you mean the biological protein involved
a patient’s narrative is a moral and clinical necessity that leads to better healing and trust. Clarification: Did you mean the biological protein involved in fungal survival, or the narrative medicine This allows farmers to turn on immunity only
Constitutively active mutants of TAR1a cause "dwarf" phenotypes due to unchecked cell death. This autoimmunity is a major barrier to yield. By studying how TAR1a remains inactive, scientists have developed "chemically inducible" NLRs—receptors that only activate in the presence of a specific agrochemical. This allows farmers to turn on immunity only when a pathogen is detected.
This polymorphism is driven by an evolutionary arms race. Pathogens mutate their effectors to avoid recognition; plants, in turn, evolve new NLR alleles. TAR1a serves as a classic model for "balanced selection" in plant populations.
The molecular steps include:
